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Marked reduced wild-type AM binding of the latex beads. Data are expressed as the mean ?SD and compared to the control in each group (WT, MS-/-, ZK1, respectively). *Significant difference compared with ZK1 (P
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Replicates following removal of recombinant sequence fragments by a blinded fully exploratory screen for recombination using RDP3. Black squares at the end of the branches represent the gag and nef sequences sampled from Cameroon in this study, while red squares represent intragene recombinant fragments in our samples. The gag tree was rooted using HIV-1 group N, O, P and SIV CPZ isolates, while t
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Dian overall survival (Figure 3). CISH analysis was possible in 44 cases.Survival probability ( )0 0 5 10 15 Months 20 25FISH EGFR = 0.2) of colorectal FISH patients showing Overall 3 (pGCN 2.6 (-------) andcancerEGFR GCN
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Hat three clones ZK1, ZK2 and ZK6 were obtained by limiting dilution and further characterized. Our PCR genotyping results verified these three clones, ZK1, ZK2 and ZK6 are MARCO-/- and SR-AI/ II-/--deficient (Fig. 1). These cell lines are able to grow rapidly in RPMI or DMEM complete media in the absence of exogenous M-CSF or other growth factors, and their doubling time is 12?6 h on the average
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C capacity functionally, but like primary AMs deficient in MARCO and SR-AI/II, ZK cells were significantly impaired in phagocytosis compared to the WT AMs due to deficiency in scavenger receptors.-Page 5 of(page number not for citation purposes)Particle and Fibre Toxicology 2008, 5:http://www.particleandfibretoxicology.com/content/5/1/A. Unopsonized Red Blood CellsNo lysisLysisB. Opsonized Red Blo
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Ogeneous cell populations. We showed here that all three of ZK cell lines responded in a manner comparable to that of primary murine alveolar macrophages. Morphologically, they are alveolar macrophage-like with Diff Quik, a modified Wright staining (Fig. 3). They all highly expressed macrophage antigens, F4/80 and CD11b on cell surfaces by immunofluorescent staining and flow cytometry assays (Fig.
1
C capacity functionally, but like primary AMs deficient in MARCO and SR-AI/II, ZK cells were significantly impaired in phagocytosis compared to the WT AMs due to deficiency in scavenger receptors.-Page 5 of(page number not for citation purposes)Particle and Fibre Toxicology 2008, 5:http://www.particleandfibretoxicology.com/content/5/1/A. Unopsonized Red Blood CellsNo lysisLysisB. Opsonized Red Blo
1
Hat three clones ZK1, ZK2 and ZK6 were obtained by limiting dilution and further characterized. Our PCR genotyping results verified these three clones, ZK1, ZK2 and ZK6 are MARCO-/- and SR-AI/ II-/--deficient (Fig. 1). These cell lines are able to grow rapidly in RPMI or DMEM complete media in the absence of exogenous M-CSF or other growth factors, and their doubling time is 12?6 h on the average

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